Bedside Topline

What It Does
Continuous or intermittent pressure, flow and oxygen-delivery measurements that show whether the pump, the filling volume or the vascular tone is failing.
Key Settings & Targets
MAP ≥65 mmHg initial shock target. Transducer at the phlebostatic axis; read PAOP at end-expiration; balloon never above the labeled volume (typically 1.5 mL on adult catheters).
Check Every Hour
  • Waveform, zero and level
  • MAP against target
  • Distal perfusion and the site
  • PA or CVP trend; balloon deflated
Top Alarm → First Move
Damped arterial trace: do not treat the number. Fast-flush test, then check air, clot, stopcocks and level.
Never
  • Titrate a vasopressor to a damped or flat trace
  • Leave the PA balloon inflated between readings or over-inflate it
  • Move a catheter with the balloon inflated
Escalate Now If
  • Hemoptysis or sudden hypoxemia during wedging (PA rupture)
  • MAP below 65 mmHg despite fluid and a vasopressor
  • Falling ScvO₂ with rising lactate
  • New limb ischemia or loss of a distal pulse

Overview & How It Works

Hemodynamic monitoring asks three questions: is the pump working, is the tank full enough, and are the pipes holding tone? It uses arterial, central venous and pulmonary artery pressures, cardiac output and derived values to answer them. The answers guide the next intervention.

Key Components

ComponentWhere It SitsWhat It Does
Arterial catheter
Radial (most often), femoral, brachial or axillary artery
Beat-to-beat pressure and mean arterial pressure (MAP); the waveform feeds pulse-pressure and variation indices
Central venous catheter (CVC)
Internal jugular, subclavian or femoral vein, tip in the superior vena cava or right atrium
Central venous pressure (CVP); with an oximetry catheter, central venous oxygen saturation (ScvO₂)
Pulmonary artery (PA) catheter
Right atrium, right ventricle, then pulmonary artery; balloon-tipped for a wedge position
Right-heart and PA pressures, pulmonary artery occlusion pressure (PAOP, also called PAWP), thermodilution cardiac output and SvO₂ if fiberoptic
Transducer and flush system
At the bedside, leveled to the phlebostatic axis
Converts pressure into an electrical signal; a pressurized continuous flush keeps the catheter open
Monitor
Bedside
Displays waveforms and calculates derived values

Flow Path

  1. Pressure is generated by the heart in whichever chamber or vessel holds the catheter tip.
  2. Pressure is transmitted along a column of fluid in the line until it reaches the transducer.
  3. The transducer converts it to an electrical signal. The monitor draws the waveform and shows the mean.
  4. Derived values come from the arithmetic, not from the transducer. Cardiac output, SVR and oxygen delivery are worked out from measured pressures, cardiac output and hemoglobin.
Key Point: A number is only as good as the waveform and the zero it sits on. Check the trace, the level and the zero before you titrate to any value.

Physiology & Core Concepts

Think of the circulation as three parts. The pump is cardiac output. The tank is preload, the filling volume and compliance of the venous system. The pipes are vascular resistance. MAP − CVP = CO × SVR ÷ 80. The same MAP can come from high flow through open pipes or from low flow through clamped ones.

What It Does

ParameterEffectWhy It Matters
Preload (venous return)
↑ stroke volume until the Frank-Starling plateau, then little further gain
Filling pressures (CVP, PAOP) can rise before stroke volume does, so high numbers do not prove the heart is full
Contractility
↑ CO at any given filling; ↓ filling pressures when the heart is strong
Failing contractility is the cardiogenic pattern: CO falls while PAOP rises
Afterload (SVR)
↑ SVR raises MAP but reduces stroke volume in a weak ventricle
Vasoconstriction helps a low-pressure patient only if the pump can still eject
Heart rate
↑ CO until diastolic filling time shortens; ↓ stroke volume at very high or very low rates
CO = HR × SV; a normal-looking CO can hide a low stroke volume
Intrathoracic pressure (PEEP, positive-pressure breaths)
↑ CVP and PAOP without more volume; ↓ venous return
Read at end-expiration or the numbers mislead
Hemoglobin and SaO₂
↓ oxygen delivery at any given CO
A falling ScvO₂ with normal CO may be anemia or hypoxemia rather than a failing pump

What It Depends On

  • In shock, preload, afterload and contractility often shift together.
  • Rhythm: losing atrial contraction or running a rapid atrial fibrillation rate both lower stroke volume.
  • Valve and wall function: regurgitation, stenosis and ischemia all change how pressures reflect volume.
  • Device position: transducer level, catheter tip position and lung volume
Note: CVP is a pressure, not a volume. Venous return, right ventricular compliance and intrathoracic pressure all show up in that one number.

Lines, Catheters & Equipment

Every access route asks a different question. Begin with the least invasive measurement that still answers yours.

TypeAccess and ConfigurationWhat It MeasuresBest ForKey Limitation or Risk
Arterial line
Radial (most often), femoral, brachial or axillary
Beat-to-beat pressure and MAP; waveform for pulse-pressure variation
Continuous MAP during vasoactive therapy; frequent blood gases
Distal ischemia; damped or inaccurate waveform
Central venous catheter
Internal jugular, subclavian or femoral vein
CVP; ScvO₂ with an oximetry catheter
Vasopressor and inotrope infusions; central access
Pneumothorax; bloodstream infection; CVP is a poor guide to volume alone
Pulmonary artery catheter
Introducer in an internal jugular, subclavian or antecubital vein
RA, RV, PA and PAOP pressures; thermodilution CO; SvO₂ if fiberoptic
Complex shock or pulmonary hypertension when other tools do not answer the question
PA rupture; arrhythmia; no mortality benefit in routine use (PAC-Man and Richard trials; FACTT in ARDS)
Pulse-contour or less-invasive CO
Uses an existing arterial line (calibrated or uncalibrated)
CO, stroke volume and variation indices
Trends when a PA catheter is not in place
Accuracy depends on the device and on the arterial waveform; follow the device IFU
Bedside ultrasound or echo
External probe
Ventricular and valve function, IVC, pericardium
Checking the interpretation of every invasive number
Operator dependent; does not replace the waveform

Pulmonary Artery Catheter Ports

  • Distal lumen: PA pressure and the PAOP wedge trace when the balloon is inflated
  • Proximal (RA) lumen: CVP, and the injectate for thermodilution
  • Thermistor near the tip: temperature change for thermodilution CO
  • Thermal filament: continuous CO on catheters that have one
  • Balloon inflation valve: the labeled maximum volume is printed on the catheter
  • Optional connectors: fiberoptic SvO₂ and a pacing lumen, when fitted
Clinical Pearl: Before you trust any PA reading, find out which lumen is hooked to the transducer. A PA tracing on the RA lumen, or an RA tracing on the distal port, means someone is reading the wrong port.

Indications, Contraindications & Evidence

Indications

  • Arterial line: vasoactive therapy that you titrate to a MAP target (initial shock target ≥65 mmHg per SSC 2021), or frequent blood gas sampling. SSC 2021 suggests invasive arterial pressure monitoring as soon as practical in septic shock, when resources allow.
  • CVC: vasoactive infusions that should not run peripherally. Central venous access also serves other therapies.
  • PA catheter: selected shock or pulmonary hypertension, when CVP, PAOP, CO and SVR together will change what you do next.
  • Pulmonary hypertension: A mean PAP above 20 mmHg is the definition. PVR above 2 Wood units points to a precapillary component (per the 2022 ESC/ERS guideline).

Contraindications

Absolute (site-specific)Relative (weigh the benefit)
Infection at the planned insertion site
Coagulopathy or severe thrombocytopenia
Absent or inadequate collateral flow for the planned arterial site
Left bundle branch block (risk of heart block during PA passage)
Thrombosis of the target vein
Tricuspid or pulmonic valve prosthesis; right-sided endocarditis
Limb whose perfusion cannot be checked
Pulmonary artery aneurysm; severe pulmonary hypertension
—
Right-heart thrombus or mass; recently implanted pacing or defibrillator leads
—
Heparin-induced thrombocytopenia: no heparin flush and no heparin-bonded catheter

Limitations

  • PAOP is not LVEDP in mitral disease, pulmonary venous obstruction, a stiff ventricle, high PEEP or severe ARDS
  • CVP does not reliably predict fluid responsiveness. Read it against the trend and the rest of the data.
  • Thermodilution CO gets less reliable with significant tricuspid regurgitation, an intracardiac shunt or an irregular rhythm.

Key Evidence

Trial or Guideline (Year)PopulationBottom Line
Richard 2003 (French PAC trial); PAC-Man 2005 (landmark trial); FACTT 2006 (landmark trial)
Richard: shock, ARDS or both. PAC-Man: general ICU patients. FACTT: ARDS, 1,000 patients
Routine or early PAC use did not improve mortality (Richard, PAC-Man). PAC-guided and central-venous-catheter-guided ARDS care had similar 60-day mortality (FACTT: 27.4% vs 26.3%)
ESCAPE 2005 (landmark trial)
Severe, recurrent heart failure
PAC guidance did not improve days alive out of hospital and increased in-hospital adverse events
ProCESS 2014 (landmark trial)
Early septic shock
Protocolized care with an ScvO₂ goal was no better for mortality than protocolized standard care or usual care; the 2001 ScvO₂ ≥70% target is historical
SSC 2021 (guideline)
Sepsis-induced hypoperfusion or septic shock
Initial MAP target 65 mmHg; dynamic measures preferred over static measures for fluid decisions
SCAI SHOCK update (2022, consensus); ACC concise guidance (2025)
Cardiogenic shock
Stages A to E describe severity; use the stage to communicate and escalate. The 2025 ACC statement is current US consensus on evaluation and management
Warning: Team review comes before insertion, even when the site is otherwise appropriate, if the patient has a tricuspid or pulmonic valve prosthesis or a left bundle branch block.

Setup, Leveling & Normal Values

The targets below are typical adult values. Each unit sets its own, so follow institutional protocol. Units are in every cell.

ParameterTypical Target / NormalIf BelowIf Above
MAP
≥65 mmHg initial shock target per SSC 2021 (a minimum, not a normal value); normal about 70–100 mmHg (typical)
Treat the cause; assess fluid responsiveness; start a vasopressor per protocol
Reassess vasopressor dose; do not treat the number alone
CVP / RAP
2–6 mmHg (typical), read at end-expiration
Low right-sided filling; interpret with the other data
RV failure, tamponade, tricuspid regurgitation, high PEEP or volume overload
Mean PAP
About 15 mmHg; pulmonary hypertension is mean PAP above 20 mmHg per the 2022 ESC/ERS guideline
—
Pulmonary hypertension: check PVR and the cause
RV and PA systolic / diastolic
RV 15–25 / 0–8 mmHg; PA 15–25 / 8–15 mmHg (typical; varies by lab)
—
High: pulmonary hypertension or RV failure; unexpected: tracing on the wrong lumen
PAOP / PAWP
6–12 mmHg (typical); above 15 mmHg suggests raised left-sided pressure per the 2022 ESC/ERS guideline
Low left-sided filling; often low preload
Raised left-sided pressure, mitral disease, or a wedge reading that is not a true PAOP
Cardiac output (CO)
4–8 L/min (typical)
Use CI for comparison across body sizes
Use the trend and the CI
Cardiac index (CI)
2.5–4.0 L/min/m² (typical)
Below 2.2 L/min/m² is commonly called low output
Interpret with SVR and filling pressures
SvO₂ / ScvO₂
SvO₂ 65–75%; ScvO₂ about 70% (typical); no fixed goal
More oxygen extraction: check hemoglobin, SaO₂ and CO
Reduced extraction or a sampling error: check the sample site

The 70% ScvO₂ target comes from the 2001 early goal-directed therapy trial, and it is historical. ProCESS found no mortality benefit from protocolized ScvO₂ care. Treat ScvO₂ as a trend.

Initial Settings

  • Leveling: transducer at the phlebostatic axis (4th intercostal space, mid-axillary line); re-level after every position change
  • Zeroing: open the stopcock to air, zero, then turn it to the patient; re-zero whenever the numbers do not fit
  • Damping (fast-flush test): One or two oscillations, then a quick return to baseline, means the damping is adequate.
  • Reading: invasive pressures at end-expiration on the ventilator; average the trace over the breathing cycle if the patient breathes spontaneously
  • Wedge: use the smallest balloon volume that gives a wedge trace, never above the volume printed on the catheter (typically 1.5 mL on adult catheters; the printed label governs), and keep each wedge brief
  • Flush system: pressurized continuous flush through a pressure bag (commonly 300 mmHg) at a set rate (commonly 3 mL/hr) (typical; follow institutional protocol)

Sizing & Positioning

Check collateral flow before you pick the arterial site. Then confirm a waveform before you trust any number. Insertion depth for central and PA catheters depends on the site and the manufacturer's markings. Position the patient for the measurement you plan, and document the transducer level.

Mean arterial pressure
MAP ≈ (SBP + 2 × DBP) ÷ 3 (estimate; the monitor integrates the waveform)

Derived Values & Shock Profiles

These derived values turn pressures into flow, resistance and oxygen delivery. Run the math only once the waveforms and zeros look right.

Systemic vascular resistance
SVR (dyn·s/cm⁵) = (MAP − CVP) × 80 ÷ CO
Pulmonary vascular resistance
PVR (dyn·s/cm⁵) = (mPAP − PAOP) × 80 ÷ CO
Oxygen delivery
DO₂ (mL O₂/min) = CO (L/min) × CaO₂ (mL O₂/dL) × 10; CaO₂ = (1.34 × Hb × SaO₂) + (0.003 × PaO₂)
VariableFormulaNormalWhy You Care
Stroke volume (SV)
CO ÷ HR
60–100 mL (typical)
Low SV with high filling pressures = pump failure; low SV with low filling = volume loss
Stroke volume index (SVI)
SV ÷ BSA
Use the trend
Indexes stroke volume for body size
Cardiac index (CI)
CO ÷ BSA
2.5–4.0 L/min/m² (see Setup)
Main output target in shock resuscitation
SVR
(MAP − CVP) × 80 ÷ CO
800–1,200 dyn·s/cm⁵ (typical)
High SVR = vasoconstriction; low SVR = distributive shock or vasoplegia
SVR index (SVRI)
(MAP − CVP) × 80 ÷ CI
Use the trend
Compare with CI to separate flow from resistance
PVR
(mPAP − PAOP) × 80 ÷ CO
Below 2 Wood units (under 160 dyn·s/cm⁵)
Above 2 Wood units = precapillary component per the 2022 ESC/ERS guideline
Oxygen delivery (DO₂)
CO × CaO₂ × 10
About 900–1,100 mL O₂/min (typical)
Low DO₂ from low CO, anemia or hypoxemia; explains low SvO₂ and rising lactate
Oxygen consumption (VO₂)
CO × (CaO₂ − CvO₂) × 10
About 200–250 mL O₂/min (typical)
With DO₂, shows whether demand or supply is the problem
Oxygen extraction ratio (O₂ER)
(SaO₂ − SvO₂) ÷ SaO₂
About 25% (typical)
Rising O₂ER means tissues are extracting more to meet demand
Cardiac power output (CPO)
MAP × CO ÷ 451 (W)
Use the trend
Pump power; falls when pressure and flow both fall
PA pulsatility index (PAPi)
(PASP − PADP) ÷ RAP
Use the trend
Lower values suggest limited right ventricular reserve; no validated cutoff for general use

Dynamic Fluid Responsiveness

  • Static pressures (CVP, PAOP) predict poorly whether a fluid bolus will raise output. SSC 2021 prefers dynamic measures for fluid decisions.
  • Pulse-pressure or stroke-volume variation can be read only in controlled mechanical ventilation, with a regular rhythm and no spontaneous breaths. Check the monitor's stated requirements.
  • Passive leg raise is a bedside test. A CO rise of 10% or more during the test predicts fluid responsiveness (a 2016 meta-analysis of 21 studies found sensitivity 0.85 and specificity 0.91). Confirm the threshold and measurement method with your protocol.
Clinical Pearl: Worked example: CO 4.0 L/min, Hb 10 g/dL, SaO₂ 96%, PaO₂ 90 mmHg. Plug those in and CaO₂ comes out at about 13.1 mL O₂/dL, with DO₂ near 525 mL O₂/min. Roughly half the typical value, and the CO still looks acceptable.

Shock Profiles at a Glance

Shock TypeCVPPAOPCISVRPrimary Fix
Hypovolemic
Low
Low
Low
High
Volume if fluid-responsive; vasopressor only as a bridge
Cardiogenic (SCAI C to E)
High
High
Low
High
Inotrope ± vasopressor; reduce afterload if MAP allows; mechanical support per team
Obstructive: massive PE
High
Low to normal
Low
High
Relieve obstruction; support the RV; avoid excess fluid
Obstructive: tamponade
High, equal to PAOP
Equal to CVP
Low
High
Pericardial drainage; fluid only as a bridge
Distributive: sepsis
Low to normal
Low to normal
High early, low later
Low
Fluid per dynamic assessment, then norepinephrine; add vasopressin when norepinephrine reaches about 0.25–0.5 mcg/kg/min per SSC 2021
Distributive: late sepsis (septic cardiomyopathy)
Variable; often high after resuscitation
Variable; may be high
Low
Variable
If CI stays low with adequate MAP: add dobutamine to norepinephrine, or use epinephrine alone per SSC 2021; reassess fluid balance
Neurogenic
Low to normal
Low to normal
Low to normal (low if bradycardic)
Low
Vasopressor to restore SVR; cautious fluid; treat bradycardia if CO falls
Key Point: Mixed shock is common. The profile names the dominant physiology. Reassess after every change rather than classifying once.

Waveforms & Console Interpretation

Normal

  • Arterial: a sharp upstroke and a dicrotic notch on the downslope. The MAP on screen is the integrated mean.
  • Right atrium: low-amplitude trace with a, c and v waves and x and y descents
  • Right ventricle: tall systolic peaks with near-zero diastolic pressure and no dicrotic notch
  • Pulmonary artery: systolic peak with a clear dicrotic notch; diastolic pressure above the wedge value
  • Wedge (PAOP): small, venous-looking trace with a mean at or slightly below the PA diastolic pressure

Abnormal Patterns

PatternWhat You SeeCauseFix
Damped arterial trace
Flattened, rounded upstroke; narrow pulse pressure
Clot or air in the line, closed stopcock, kinked tubing, catheter against the vessel wall
Fast-flush test; clear the line; re-level and re-zero; reposition per protocol
Under-damped trace
Extra high-frequency oscillations; overshoot on the systolic peak
Short, stiff tubing or a poorly set-up line
Re-check the set-up per the manufacturer; repeat the fast-flush test
Spontaneous wedge
PAOP-type trace on the distal lumen with the balloon deflated
Catheter has migrated into a smaller branch
Keep the balloon deflated; confirm position on chest X-ray; reposition per protocol
RV pattern on the PA lumen
Tall systolic waves with near-zero diastolic pressure
Catheter has returned to the right ventricle
Withdraw per protocol and confirm position; watch for ectopy while withdrawing
Over-wedge or prolonged wedge
Wedge trace persists after deflation; PAOP above PA diastolic
Balloon volume too large or wedge held too long
Deflate; re-check; use the smallest volume and a brief wedge
Respiratory swings
Large cyclic changes in CVP or PAOP
High PEEP or positive-pressure breaths
Read at end-expiration; average the trace
Prominent x descent, absent y descent
Flattened venous trace with a steep x descent
Cardiac tamponade (equalized diastolic pressures)
Urgent team review; pericardial drainage per protocol
Giant v waves
Large v wave on the RA or wedge trace
Tricuspid regurgitation or mitral regurgitation
Interpret with echo before acting on the number
Warning: With the balloon deflated, a PA-type trace on the distal lumen is a catheter position problem until proven otherwise. Do not chart it as a PAOP.

Monitoring & Ongoing Care

At the bedside, monitoring is routine. The waveform, the site and the trend matter more than any one number.

FrequencyWhat to CheckExpected / Action Threshold
Hourly
Waveform quality, zero and transducer level; MAP against target; PA or CVP trend; distal perfusion, capillary refill time as an adjunct per SSC 2021, and the site
MAP at or above target (≥65 mmHg initial shock target); a damped or lost trace means fix the system first
Every 4–6 h
CO and CI with each intervention; SvO₂ or ScvO₂ trend; blood gases and lactate per protocol; urine output
Trend in the right direction after each change; rising lactate or falling ScvO₂ prompts review
Every 12–24 h
Line need and dressing; CBC; electrolytes including K⁺, Mg and iCa; creatinine; chest X-ray for catheter position
Remove any line that no longer changes management
Daily
Indication review; complications screen (bleeding, infection, limb); team plan for each catheter
Document the reason each catheter stays in

Positioning & Mobility

  • Keep the transducer leveled to the phlebostatic axis whenever the patient moves; re-zero after any change
  • Secure lines and tubing at every position change, and keep the arterial limb in view
  • Head-of-bed position and hip-flexion limits follow the unit protocol and the catheter site
Clinical Pearl: Trace the line from the catheter to the transducer at every shift. Clots, air, closed stopcocks and kinks cause most lost waveforms, not a failing heart.

Key Medications & Drug Considerations

Doses vary by protocol; follow local protocols and medical direction. Infusion rates are weight-based unless stated. Check the units on the pump before you start.

MedicationRole HereTypical Adult Dose / TargetDevice-Specific Notes
Norepinephrine
First-line vasopressor per SSC 2021
0.05–0.5 mcg/kg/min IV infusion, titrated to MAP ≥65 mmHg High-alert
Confirm mcg/kg/min, not mcg/min. SSC 2021 suggests starting peripherally rather than delaying for central access: use a vein in or proximal to the antecubital fossa, for a short period only, and watch for extravasation; move to central access when feasible
Vasopressin
Add-on for septic shock: SSC 2021 suggests adding it instead of escalating norepinephrine; its remarks say it is usually started when norepinephrine reaches about 0.25–0.5 mcg/kg/min (a practice remark, not a fixed trigger)
0.03 units/min IV infusion, fixed dose High-alert
Units per minute, not units per hour
Dobutamine
Inotrope for low CI with adequate MAP
Start 0.5–1 mcg/kg/min IV infusion; usual 2–20 mcg/kg/min High-alert
Can lower BP and cause tachyarrhythmia; run with a vasopressor if MAP is low
Milrinone
Inodilator for selected low-output states
No load in shock; start 0.125–0.25 mcg/kg/min IV infusion; usual 0.125–0.5; maximum 0.75 mcg/kg/min High-alert
Vasodilation can lower MAP; lower the dose for renal impairment
Heparin (flush solution)
Keeps arterial and PA lines patent
Per institutional protocol; concentration varies High-alert
Label every flush bag with drug and concentration. Avoid heparin flush and heparin-bonded catheters if HIT is suspected or confirmed; report a platelet fall

Pattern to Drug

PatternCVP / PAOPCI / SVRFirst Choice and Add-On
Low output, high resistance, MAP adequate (cardiogenic pattern)
High / high
Low / high
Dobutamine; add norepinephrine if MAP falls; mechanical support per team
Low output, low resistance (MAP low)
Low to normal / low to normal
Low / low
Fluid if responsive; norepinephrine; add dobutamine if CI stays low after MAP is restored
Normal or high output, low resistance (distributive)
Low to normal / low to normal
High to normal / low
Fluid per dynamic assessment; norepinephrine; add vasopressin when norepinephrine reaches about 0.25–0.5 mcg/kg/min per SSC 2021
High pressure, low flow, high PVR (RV failure or PE)
High / normal to low
Low / high
Relieve the cause; support the RV; avoid excess fluid; vasoactive choice per team
Tamponade
High, equalized
Low / high
Pericardial drainage; fluid only as a bridge; avoid vasodilators

Pediatric doses are in Special Populations. For more dosing detail, use the linked medication pages.

Alarms & Troubleshooting

Look at the patient first and the machine second. Before you blame the monitor, confirm the patient is perfused.

Alarm or ProblemWhat You SeeLikely CausesFirst Actions
Damped arterial trace
Flattened or rounded upstroke; narrow pulse pressure
Clot or air in line; closed or partly closed stopcock; kinked tubing; catheter against the wall
1) Check the patient, MAP and limb 2) Fast-flush test 3) Trace the line for air, clot and stopcocks 4) Re-level and re-zero 5) Reposition or replace per protocol
Lost arterial or PA waveform
Flat line or no trace
Disconnected cable or transducer; kinked or clotted line; catheter migrated against wall
1) Check the patient 2) Check cable, transducer and connections 3) Fast-flush test 4) Use a cuff MAP until fixed
Spontaneous wedge
PAOP-type trace with balloon deflated
Catheter migrated into a smaller branch
1) Keep balloon deflated 2) Notify team 3) Confirm on chest X-ray; reposition per protocol
RV pattern on PA lumen
Tall systolic waves; near-zero diastole
Catheter back in right ventricle
1) Withdraw per protocol 2) Watch for ectopy 3) Confirm position
Numbers that do not fit
PAOP above PA diastolic; CO that jumps without a change in patient
Over-wedging; respiratory swings; thermodilution technique (injectate volume or temperature); tricuspid regurgitation
1) Deflate and re-wedge with the smallest volume 2) Read at end-expiration 3) Repeat CO with consistent technique 4) Check with echo
Hemoptysis or sudden hypotension during wedge
Blood from the airway; falling SpO₂ and BP
PA rupture or pulmonary infarction
1) Stop all wedging; keep the balloon deflated unless the physician directs otherwise 2) Call for help 3) See Emergencies
Bleeding or air at a disconnected line
Blood at the site; pressure bag empty; air in the tubing
Loose connection; stopcock turned the wrong way
1) Apply direct pressure to the site 2) Close the stopcock 3) Replace or secure the connection
Never: Never titrate a vasopressor to a damped or flat trace. Keep the PA balloon deflated during routine monitoring, and never inflate it past its labeled volume. Never move a catheter while the balloon is inflated. Never accept a number you have not checked against the waveform.

Complications & Red Flags

Aseptic insertion, a brief wedge and a daily check on need prevent most complications.

ComplicationHow You Recognize ItImmediate ActionPrevention
Insertion: pneumothorax or hemothorax
Dyspnea, falling SpO₂, reduced breath sounds, rising airway pressure
Urgent chest X-ray; decompression and chest tube per protocol
Ultrasound-guided access; confirm position on imaging
Insertion: arrhythmia
Ventricular ectopy, VT or VF, or new bundle branch block during passage
Withdraw per protocol; treat per ACLS
Continuous ECG; extra caution with left bundle branch block; catheter whip in the right ventricle can cause ectopy
Insertion: cardiac perforation
Hypotension with muffled heart sounds or falling pressure during passage; pericardial effusion on echo
Stop advancing; urgent echo; pericardiocentesis or surgery per team
Advance only with the waveform in view; never force past resistance
Insertion: knotting or coiling
Resistance on passage; abnormal trace; coil on X-ray
Do not force; team-directed retrieval
Advance slowly with the waveform in view
During use: PA rupture
Hemoptysis, sudden hypoxemia, hypotension
Stop wedging; keep the balloon deflated unless the physician directs otherwise; if the bleeding side is known, position it down; protect the airway and reverse anticoagulation per team; urgent team and surgical review
Smallest volume; brief wedge; avoid wedging with high PA pressure or coagulopathy
During use: pulmonary infarction
Pleuritic pain, hemoptysis, new infiltrate
Stop wedging; evaluate for embolism
Limit wedge time; avoid repeated wedging
During use: bloodstream infection or endocarditis
Fever, rigors, site inflammation, positive cultures
Culture; remove the catheter per protocol; treat
Aseptic insertion and dressing care; remove when no longer needed
During use: limb ischemia
Pale, cool or numb hand or foot; loss of distal pulse or capillary refill
Remove the arterial catheter; warm the limb; notify team; vascular review
Check distal perfusion every hour; choose the site with collateral flow
During use: valve injury
New murmur or new regurgitation on echo; resistance on withdrawal
Do not withdraw by force; echo and team review before further movement
Limit time in the right heart; avoid forced advancement or withdrawal
During use: bleeding or air
Brisk blood at the site; air or leak at a connection
Firm direct pressure; close the stopcock; call for help
Lock all connections; keep lines capped and secured
Red Flags: Call Immediately
  • Hemoptysis with a PA catheter in place: treat it as PA rupture until proven otherwise.
  • Sudden hypoxemia or hypotension while wedging.
  • An arterial line limb that turns pale, cold, numb or pulseless.
  • Brisk bleeding, or air in a pressurized line.
  • A new ventricular arrhythmia once the catheter has passed.

Emergencies

Each scenario below starts with the life-saving action. Steps beyond EMS or general RN scope are marked, and each follows local protocol.

Hemoptysis or Sudden Shock With a PA Catheter

  1. Stop the wedge: stop all wedge measurements. Keep the balloon deflated unless the physician directs otherwise; balloon management after rupture (deflation, or re-inflation to tamponade bleeding) is a physician decision.
  2. Protect the airway: if the bleeding side is known, position the patient with that lung down. Give oxygen and support breathing per protocol. The airway team considers lung isolation (selective intubation, double-lumen tube or bronchial blocker) to protect the unaffected lung (ICU/airway team).
  3. Reverse anticoagulation (heparin and other agents) and correct coagulopathy per the team (ICU team).
  4. Call for help: notify the ICU or cardiology team and request urgent surgical review (suspected PA rupture).
  5. Treat shock per protocol while the team arrives; document the time of the first hemoptysis.
  6. Imaging and specialist review per team: CT, bronchoscopy and embolization or surgery as indicated (ICU/cardiology team).

Bleeding From an Arterial or PA Line

  1. Apply direct pressure over the puncture site above the catheter.
  2. Close the stopcock and stop the pressure bag if the pressurized line has disconnected.
  3. Call for help and check distal perfusion in the limb.
  4. Remove or reinsert the line only on team direction (ICU/CCT per protocol).

Air Embolism Through a Central Line

  1. Clamp the line at once and stop any infusion through it.
  2. Call for help and give high-flow oxygen.
  3. Position the patient per the institutional protocol for venous air embolism.
  4. Treat the consequences (hypotension, arrhythmia) per ACLS and unit protocol.

Cardiac Arrest With Monitoring Lines in Place

  1. Start CPR and defibrillation per AHA 2025 ACLS; the lines do not change compressions or shocks.
  2. Keep the catheters in place and do not stop compressions to re-zero the transducer.
  3. Use the arterial waveform if one is in place: the diastolic pressure can help judge compression quality, and a sudden rise in arterial pressure can signal return of circulation; confirm with a pulse check. Use the numeric targets in current AHA guidance and your protocol.
  4. After return of circulation, use the arterial waveform to check perfusion and guide vasoactive therapy per protocol.

Monitoring Power or Cable Failure

  1. Get a MAP from the backup: cuff pressure or a second monitor until the trace is back.
  2. Keep the line patent under the pressure bag; check for leaks and air.
  3. Call for help if the fault cannot be fixed at the bedside.

Removal & Discontinuation

Readiness to Remove

Remove each catheter once its measurement stops changing management, or once the catheter itself is causing harm. Document why it stays in.

Removal Steps

  1. Confirm the indication and get team agreement.
  2. PA catheter: deflate the balloon fully before any movement, then withdraw slowly with the waveform in view.
  3. Arterial line: hold firm pressure until hemostasis; check distal perfusion.
  4. Central line: remove per protocol; apply an occlusive dressing.
  5. Afterward: check the site and limb for bleeding and hematoma, and document.
Protocol Varies: Each institution sets its own timing, pressure-hold duration and patient position for removal. Follow local protocol.

Transport Considerations

Before You Move

  1. Power: confirm battery charge and backup power for the monitor.
  2. Flush and pressure bag: confirm the pressure bag and flush supply are working.
  3. Balloon: confirm the PA balloon is deflated and the catheter is secured.
  4. Lines: secure and label every catheter and tubing; spare transducer and cable if available.
  5. Team: brief the receiving team on the device, the last numbers and the trigger points.

If You Are Not the Device Specialist

EMS and general RN guidance

  • Watch the limb with the arterial line; report a cold, pale or numb hand or foot
  • Do not wedge, inflate or flush the PA lumen; do not move or disconnect a PA catheter
  • If an arterial line bleeds, apply direct pressure; if a line is disconnected, close the stopcock
  • If the trace is lost, report it; use the cuff for BP and call for help
  • ACLS governs CPR and defibrillation. The lines stay in place.

During Transport

  • Re-level and re-zero after every move that changes the patient's position
  • Check the pressure bag, the flush and the tubing security at every handoff
  • Watch for air in the tubing and any change in the trace

Air Medical & Altitude

Boyle's law explains why: gas in a closed line expands as ambient pressure falls. Before flight, and during ascent or descent, check every connection for air.

Handoff

  • Monitoring type, site and insertion date
  • Last MAP, CVP or PAOP, and CI with the time measured
  • Vasoactive drugs and doses, and the target MAP
  • Damping status and the last zero and level
  • Limb checks, recent alarms and complications
  • Anticoagulation and the latest labs (lactate, hemoglobin, platelets)
Transport Pearl: Make sure the PA balloon is deflated before the patient moves. Then re-level and re-zero at the new position.

Special Populations

Pediatric & Neonatal

Pediatric MAP targets and normal values depend on age and size, so do not use adult numbers for children. Arterial lines are common in pediatric intensive care. PA catheters are rarely used in children because of their size. For normal vital signs, shock and vascular access, use the pediatric pages: /pediatrics/vital-signs.html, /pediatrics/shock.html and /pediatrics/vascular-access.html. Pediatric hypotension is defined by age. Use the thresholds on the pediatric shock page, not adult values. Neonatal and infant lines run at lower flush rates than adult lines, so follow the unit's pediatric flush protocol.

Pregnancy & Peripartum

Pregnancy raises cardiac output and lowers SVR, so adult normal values can mislead you. Late in pregnancy, lying supine compresses the aorta and vena cava and lowers preload. Use left lateral tilt or uterine displacement, and hold the same position for serial readings. PA catheter use in pregnancy is uncommon. It is a team decision made with the obstetric team.

Other

Indexed values (CI, SVRI, SV index) depend on which body surface area formula you use. Keep the same formula across serial measurements.

  • Obesity: use a correctly sized cuff for noninvasive MAP, and confirm invasive readings on the waveform
  • ECMO or ventricular assist devices: the arterial waveform may be reduced or absent. Follow the device team's monitoring plan.
Note: Neonatal and pediatric normal values cannot be swapped for adult values.

Bedside Pearls & Teaching Script

Systematic Walk-Through

  1. Trust the hardware: check level, zero, damping and the waveform.
  2. Look at the pump: MAP, CI and SvO₂ or ScvO₂ against the target.
  3. Look at the tank: CVP and PAOP, and whether a fluid challenge is likely to help.
  4. Look at the pipes: SVR and PVR.
  5. Integrate: lactate, urine output, skin, mental status and echo.
  6. Decide and reassess: fluid, vasopressor, inotrope or mechanical support, then trend the result.

Pearls

  • The trend beats any single number.
  • Full and failing, with high PAOP and low CI, is not the same as empty and failing, with low PAOP and low CI.
  • Read the waveform before you read the number.
  • One MAP, many causes.
  • A catheter only answers the question someone asks it.

References

  1. Evans, L., Rhodes, A., Alhazzani, W., Antonelli, M., et al. (2021). Surviving Sepsis Campaign: International guidelines for management of sepsis and septic shock 2021. Critical Care Medicine, 49, e1063–e1143. https://doi.org/10.1097/CCM.0000000000005337
  2. Humbert, M., Kovacs, G., Hoeper, M. M., Badagliacca, R., et al. (2022). 2022 ESC/ERS Guidelines for the diagnosis and treatment of pulmonary hypertension. European Heart Journal, 43, 3618–3731. https://doi.org/10.1093/eurheartj/ehac237
  3. Naidu, S. S., Baran, D. A., Jentzer, J. C., et al. (2022). SCAI SHOCK Stage Classification Expert Consensus Update: A review and incorporation of validation studies. Journal of the American College of Cardiology, 79, 933–946. https://doi.org/10.1016/j.jacc.2022.01.018
  4. Sinha, S. S., Morrow, D. A., Kapur, N. K., et al. (2025). 2025 concise clinical guidance: An ACC expert consensus statement on the evaluation and management of cardiogenic shock. Journal of the American College of Cardiology, 85, 1618–1641. https://doi.org/10.1016/j.jacc.2025.02.018
  5. Richard, C., Warszawski, J., Anguel, N., et al. (2003). Early use of the pulmonary artery catheter and outcomes in patients with shock and acute respiratory distress syndrome: A randomized controlled trial. JAMA, 290, 2713–2720. https://doi.org/10.1001/jama.290.20.2713
  6. Harvey, S., Harrison, D. A., Singer, M., et al. (2005). Assessment of the clinical effectiveness of pulmonary artery catheters in management of patients in intensive care (PAC-Man): a randomised controlled trial (landmark trial). The Lancet, 366, 472–477. https://doi.org/10.1016/s0140-6736(05)67061-4
  7. Wheeler, A. P., Bernard, G. R., Thompson, B. T., Schoenfeld, D., Wiedemann, H. P., deBoisblanc, B., Connors, A. F., Jr., Hite, R. D., Harabin, A. L., et al. (2006). Pulmonary-artery versus central venous catheter to guide treatment of acute lung injury (FACTT; National Heart, Lung, and Blood Institute ARDS Clinical Trials Network). New England Journal of Medicine, 354, 2213–2224. https://doi.org/10.1056/NEJMoa061895
  8. Binanay, C., Califf, R. M., Hasselblad, V., et al. (2005). Evaluation study of congestive heart failure and pulmonary artery catheterization effectiveness: The ESCAPE trial (landmark trial). JAMA, 294, 1625–1633. https://doi.org/10.1001/jama.294.13.1625
  9. ProCESS Investigators, Yealy, D. M., Kellum, J. A., et al. (2014). A randomized trial of protocol-based care for early septic shock (landmark trial). New England Journal of Medicine, 370, 1683–1693. https://doi.org/10.1056/NEJMoa1401602
  10. Rivers, E., Nguyen, B., Havstad, S., et al. (2001). Early goal-directed therapy in the treatment of severe sepsis and septic shock (landmark trial). New England Journal of Medicine, 345, 1368–1377. https://doi.org/10.1056/NEJMoa010307
  11. Monnet, X., Marik, P., et al. (2016). Passive leg raising for predicting fluid responsiveness: A systematic review and meta-analysis. Intensive Care Medicine, 42, 1935–1947. https://doi.org/10.1007/s00134-015-4134-1
  12. American Heart Association. (2025). Part 9: Adult advanced life support: 2025 American Heart Association guidelines for cardiopulmonary resuscitation and emergency cardiovascular care. Circulation, 152. https://doi.org/10.1161/cir.0000000000001376

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