Bedside Snapshot

Normal (Adult)
Arterial: pH 7.35–7.45 ([H⁺] 35–45 nmol/L)
Venous: about 0.04 lower (7.31–7.41)
Critical Values
pH <7.20 or >7.60 ([H⁺] >63 or <25 nmol/L); typical, lab-specific
High Suggests
  • Hyperventilation: hypoxemia, PE, sepsis, early salicylate
  • Overventilation by bag or ventilator
  • Vomiting or NG suction
  • Diuretics, low K⁺, alkali load
Low Suggests
  • Hypoventilation: opioids, COPD, exhaustion
  • Lactic acidosis: shock, sepsis, seizures
  • DKA, including euglycemic
  • Toxic alcohols, salicylate, kidney failure
Act Now If
pH <7.20 or >7.60, PaCO₂ rising as pH falls, or altered mental status: support ventilation, monitor and 12-lead ECG, check K⁺, glucose and ionized Ca²⁺; notify receiving/medical control
Watch Out For
A normal pH does not mean a normal acid–base state: two opposing disorders can cancel out. Read PaCO₂, HCO₃⁻ and the anion gap every time.

Reference & Critical Ranges

Lab-specific ranges: Every lab sets its own range, and ranges shift with the analyzer and the population. Use the range on your lab report. The values below are typical.

Reference ranges

PopulationConventionalSINotes
Adult: arterial
pH 7.35–7.45
[H⁺] 35–45 nmol/L
Mean about 7.40 ([H⁺] 40 nmol/L)
Adult: peripheral venous
pH 7.31–7.41
[H⁺] about 39–49 nmol/L
About 0.04 lower than arterial (Critical Care Transport); read against venous norms
Pregnancy
Upper end of normal (third-trimester mean about 7.44)
[H⁺] about 36 nmol/L
Compensated respiratory alkalosis: PaCO₂ near 30 mmHg, HCO₃⁻ lower
Older adult
Same as adult
Same as adult
—
Children
Same as adult
Same as adult
Neonates: use the lab's age-specific range; cord and first-day values differ

Severity & critical values

LevelConventionalSIWhat it means / typical findings
Critical (low)
pH <7.20
[H⁺] >63 nmol/L
Severe acidemia: falling contractility and BP, poor vasopressor response, dysrhythmia. Below 7.00 ([H⁺] >100 nmol/L) is the severe DKA band
Acidemia
pH 7.20–7.34
[H⁺] 46–63 nmol/L
Find the cause: PaCO₂ high (respiratory) or HCO₃⁻ low (metabolic). DKA criterion is pH <7.30
Normal
pH 7.35–7.45
[H⁺] 35–45 nmol/L
Does not exclude a mixed disorder: check PaCO₂, HCO₃⁻ and the anion gap
Alkalemia
pH 7.46–7.60
[H⁺] 25–34 nmol/L
Hyperventilation or metabolic alkalosis; expect low K⁺ and ionized Ca²⁺
Critical (high)
pH >7.60
[H⁺] <25 nmol/L
Severe alkalemia: low ionized Ca²⁺ with tetany, seizures, delirium; dysrhythmia; arteriolar and coronary constriction
Critical values: pH <7.20 or >7.60 ([H⁺] >63 or <25 nmol/L) marks the critical lines. These are typical, and your lab sets its own. Support ventilation, get the patient on a monitor, draw K⁺, glucose and ionized Ca²⁺, and call the prescriber or medical control now.

Conversion: [H⁺] in nmol/L = 10(9 − pH). pH 7.40 gives 40 nmol/L. Every 0.1 drop in pH adds about 25% to [H⁺].

Henderson–Hasselbalch equation
pH = 6.1 + log₁₀ [HCO₃⁻ ÷ (0.03 × PaCO₂)]

Plug in normal values: HCO₃⁻ 24 mEq/L and PaCO₂ 40 mmHg give a 20:1 ratio and a pH of 7.40. The analyzer works out HCO₃⁻ from the measured pH and PaCO₂ using this equation.

Bedside [H⁺] check (Kassirer–Bleich)
[H⁺] (nmol/L) = 24 × PaCO₂ ÷ HCO₃⁻

Try it at normal values: 24 × 40 ÷ 24 = 40 nmol/L (pH 7.40). When the answer and the reported pH disagree, suspect a transcription or sampling error.

Causes of Abnormal Results

Low (acidemia, pH <7.35)

Ask which value pulled the pH down. Was it a high PaCO₂ (respiratory acidosis) or a low HCO₃⁻ (metabolic acidosis)? Both at once is the most dangerous pattern.

MechanismCommon causesBedside clue
Respiratory: low drive
Opioids, sedatives, alcohol, head injury, stroke
Slow shallow breathing, rising EtCO₂, pinpoint pupils
Respiratory: lungs and airway
COPD exacerbation, tiring asthma, pneumonia, pulmonary edema, airway obstruction
Wheeze, accessory muscles, fatigue, confusion
Respiratory: pump failure
Guillain–Barré, myasthenia, high spinal cord injury, flail chest, obesity hypoventilation
Weak cough, paradoxical breathing, falling vital capacity
Metabolic, high anion gap: lactate
Shock, sepsis, hypoxia, seizures, CO or cyanide, metformin with AKI
Poor perfusion, high lactate
Metabolic, high anion gap: ketoacids
DKA (including euglycemic on SGLT2 inhibitors), alcoholic, starvation
Kussmaul breathing, vomiting, ketones
Metabolic, high anion gap: toxins and kidney
Methanol, ethylene glycol, salicylate; advanced AKI or CKD
Osmolal gap; tinnitus; rising creatinine
Metabolic, normal anion gap
Diarrhea, large-volume 0.9% saline, renal tubular acidosis, acetazolamide
High Cl⁻

High-anion-gap mnemonic (GOLDMARK): Glycols, Oxoproline, L-lactate, D-lactate, Methanol, Aspirin, Renal failure, Ketoacidosis. MUDPILES is the older version. The anion gap and delta gap formulas are on the bicarbonate page.

Mixed acidemia falls fastest: Metabolic acidosis plus hypoventilation drops the pH far more than either one alone. Think of the tiring DKA patient, the septic patient, the sedated patient, or the post-arrest patient on a low ventilator rate. Check whether the PaCO₂ sits above what Winter's formula predicts.

High (alkalemia, pH >7.45)

Is the PaCO₂ low (respiratory alkalosis) or the HCO₃⁻ high (metabolic alkalosis)? Metabolic alkalosis is the most common acid–base disorder in ICU patients. Treatment is often the cause.

MechanismCommon causesBedside clue
Respiratory: hypoxemia and lung
Pulmonary embolism, pneumonia, pulmonary edema, altitude
Tachypnea, low SpO₂ (PE may have a normal SpO₂)
Respiratory: central drive
Sepsis, early salicylate toxicity, stroke, fever, pain, anxiety, pregnancy
Tachypnea out of proportion; paresthesias
Respiratory: iatrogenic
Bag-valve-mask or ventilator overventilation
Low EtCO₂ on the monitor
Metabolic: acid loss
Vomiting, NG suction
Low Cl⁻ and K⁺
Metabolic: kidney and volume
Loop or thiazide diuretics, mineralocorticoid excess, low K⁺ or Mg²⁺
Low K⁺; hypertension if mineralocorticoid
Metabolic: alkali or posthypercapnic
Sodium bicarbonate, citrate in massive transfusion; chronic CO₂ retainer ventilated down fast
Alkalemia after treatment or intubation
Hyperventilation is a finding, not a diagnosis: Before you call it anxiety, rule out hypoxemia, PE, sepsis, salicylate toxicity and compensation for metabolic acidosis. Never have a patient rebreathe into a bag. It can cause hypoxia and blunt compensation for an unrecognized metabolic acidosis.

Interpretation & Bedside Approach

Step-by-step approach

  1. Is it real? Arterial or venous? Air bubbles, delay or a line sample with flush can all move the result. If pH, PaCO₂ and HCO₃⁻ fail the [H⁺] check, repeat the sample.
  2. How sick is the patient? Check mental status, breathing rate and depth, EtCO₂, perfusion, ECG and K⁺. A tiring patient can show a normal-looking pH and still be minutes from arrest.
  3. Read the pH first. Under 7.35 is acidemia. Over 7.45 is alkalemia. The primary disorder is the one that pushes the pH in that direction.
  4. Find the cause in PaCO₂ and HCO₃⁻. PaCO₂ moving opposite the pH points to respiratory. HCO₃⁻ moving with the pH points to metabolic. Acute or chronic? Compare with the patient's baseline. The trend tells you more than any single value.
  5. Check compensation and derived values. Use the table below. Compensation that falls short or overshoots means a second disorder. In any metabolic acidosis, calculate the albumin-corrected anion gap and delta gap.
  6. Name it and escalate. By the 2024 ADA/EASD/JBDS/AACE/DTS consensus, DKA is pH <7.30 and/or HCO₃⁻ <18 mEq/L with ketonemia. It is severe at pH <7.00. In a COPD exacerbation, the GOLD 2026 report treats PaCO₂ >45 mmHg with pH <7.35 as acute hypercapnic respiratory failure. Acidosis to pH ≤7.25 with PaCO₂ >60 mmHg or above baseline marks ventilatory failure (Rome classification).

Expected compensation

Primary disorderPrimary changeExpected compensation
Metabolic acidosis
↓ HCO₃⁻
PaCO₂ = (1.5 × HCO₃⁻) + 8 ± 2 mmHg (Winter's formula)
Metabolic alkalosis
↑ HCO₃⁻
PaCO₂ rises 0.6–0.75 mmHg per 1 mEq/L rise; not above 55 mmHg from compensation alone
Respiratory acidosis, acute
↑ PaCO₂
HCO₃⁻ rises 1–2 mEq/L per 10 mmHg rise
Respiratory acidosis, chronic (3–5 days)
↑ PaCO₂
HCO₃⁻ rises 3–4 mEq/L per 10 mmHg rise
Respiratory alkalosis, acute
↓ PaCO₂
HCO₃⁻ falls 1–2 mEq/L per 10 mmHg fall
Respiratory alkalosis, chronic (2–3 days)
↓ PaCO₂
HCO₃⁻ falls 4–5 mEq/L per 10 mmHg fall

Compensation brings the pH back toward normal, but only partway. It never overshoots.

Patterns

PatternSuggests
pH low, PaCO₂ high, HCO₃⁻ normal or slightly high
Acute respiratory acidosis (opioids, tiring asthma)
pH low, HCO₃⁻ low, PaCO₂ low and within Winter's range
Compensated metabolic acidosis
pH low, HCO₃⁻ low, PaCO₂ normal or high
Metabolic plus respiratory acidosis: ventilation is failing
pH high, PaCO₂ low, HCO₃⁻ low
Respiratory alkalosis; HCO₃⁻ lower than predicted means an added metabolic acidosis (sepsis, salicylate)
pH near normal, PaCO₂ and HCO₃⁻ both abnormal
Mixed disorder until proven otherwise
pH, PaCO₂ and HCO₃⁻ normal, anion gap high
Hidden high-gap acidosis offset by metabolic alkalosis (DKA with vomiting)
Worked example: pH 7.40, PaCO₂ 60 mmHg, HCO₃⁻ 36 mEq/L. Chronic CO₂ retention should bring the HCO₃⁻ to about 30–32 mEq/L, and full compensation would leave the pH below normal. At 36, the HCO₃⁻ is higher than that. The picture is chronic respiratory acidosis plus a metabolic alkalosis (diuretics, vomiting, steroids), not full compensation.
Acid-base nomogram plotting arterial pH and [H+] against plasma bicarbonate with PaCO2 isobars and disorder bands
Figure 1: Acid–base nomogram. Arterial pH (bottom axis) and [H⁺] (top axis) against plasma HCO₃⁻, with PaCO₂ isobars and a band for each simple disorder. A result outside every band suggests a mixed disorder. Source: Wikimedia Commons, Huckfinne, public domain

Management Priorities

Emergency: Severe acidemia (pH <7.20)

  1. Ventilation first if PaCO₂ is high: airway, bag-valve-mask, naloxone for opioid toxicity, and NIV for a COPD exacerbation (the initial mode for acute hypercapnic failure). Intubate if NIV fails or is contraindicated (Paramedic/RN/CCT, per local protocol).
  2. Protect compensation if PaCO₂ is low: Do not oversedate. If intubation is unavoidable, keep apnea as brief as you can and set a high minute ventilation right away, matched to the pre-intubation PaCO₂ or EtCO₂.
  3. Treat the cause: Restore perfusion and control the source in shock or sepsis. Give fluids, insulin and K⁺ for DKA. For toxic alcohols or salicylate, involve the poison center and plan for dialysis.
  4. Watch K⁺ and glucose: Get the ECG, K⁺ and glucose now, and keep checking them. Treat hyperkalemia directly. Do not wait for the pH to be corrected before you lower it.
  5. Bicarbonate only for a specific reason: the table lists the common indications, but it is not the full list. Sodium-channel-blocker toxicity with wide QRS or arrhythmia, and hyperkalemia with ECG changes, are also indications, per poison center and protocol. Do not use it to raise blood pressure in lactic acidosis, routinely in cardiac arrest, or (almost never) for respiratory acidosis. Salicylate toxicity calls for alkalinization under toxicology direction.

Emergency: Severe alkalemia (pH >7.60)

  1. Monitor: Put the patient on an ECG. Check K⁺, ionized Ca²⁺, Mg²⁺ and phosphate. Start seizure precautions.
  2. Respiratory cause: bring the bag or ventilator rate or tidal volume down toward the patient's baseline PaCO₂. Treat hypoxemia, pain and the cause (PE, sepsis, salicylate).
  3. Metabolic cause: Where possible, halt alkali, citrate and diuretics. For chloride-responsive alkalosis (vomiting, NG loss), give 0.9% saline with K⁺. Replace K⁺ and Mg²⁺.
  4. Refractory: ICU care only: acetazolamide, or dialysis. Dilute hydrochloric acid (0.1–0.2 N, 0.1–0.2 mmol/kg/h) is the last line. Pharmacy prepares it as an infusion for pH >7.6 when dialysis is not possible. It has to go through a central catheter, with frequent blood gas and electrolyte checks, per institutional protocol.

Medications

Doses vary by protocol; follow local protocols and medical direction.

DrugAdult dosePediatric dose (max)Onset / key notes
Sodium bicarbonate: septic shock with severe metabolic acidemia
pH ≤7.20 with AKI stage 2–3: IV infusion per ICU protocol, titrated to pH (titrate to effect: no fixed max)
Not routinely used (consult)
SSC 2021 suggests it here; recommends against it to improve hemodynamics in lactic acidosis. Ventilation must clear the extra CO₂; watch Na⁺, ionized Ca²⁺
Sodium bicarbonate: DKA
pH <7.0 only, per DKA protocol (titrate to pH >7.0: no fixed max)
Not routinely used (consult)
Fluids and insulin clear DKA acidosis; bicarbonate adds hypokalemia risk and is linked to cerebral injury in children
Naloxone: opioid hypoventilation
0.2–2 mg IV/IO/IM or 2–4 mg IN, every 2–3 min, titrated to breathing (no fixed max). In opioid-dependent patients give cautiously in small increments: abrupt full reversal can precipitate acute withdrawal
0.01 mg/kg IV/IO initial dose (label), then 0.1 mg/kg for later doses if no response, every 2–3 min; protocol-dependent. Use the low end for opioid-dependent or chronically exposed children (risk of acute withdrawal and pulmonary edema). No fixed max in the label
IV onset within 2 min. Ventilate first; titrate to breathing, not alertness; may wear off before the opioid
Sodium bicarbonate: salicylate toxicity
Alkalinizing infusion per toxicology protocol (urine alkalinization, after volume and electrolyte correction)
Per toxicology protocol
Potassium chloride is added to support urinary alkalinization; keep K⁺ replete and watch for fluid overload. Avoid drugs that depress respiratory drive, which blunt the hyperventilation holding up the pH
Acetazolamide: metabolic alkalosis
250–375 mg IV or PO once daily (label edema dose). Off-label for metabolic alkalosis; dose per ICU protocol
Not routinely used (consult)
Increases HCO₃⁻ excretion; for posthypercapnic alkalosis or when saline is unsuitable (volume overload). Raises urine K⁺ and phosphate losses: recheck them. Avoid with hypokalemia, hyponatremia, sulfonamide allergy or significant hepatic or renal failure. Correct K⁺ first

Definitive care & disposition

  • ICU: pH <7.20 or >7.60, rising PaCO₂ despite NIV, severe DKA (pH <7.00), any bicarbonate or acid infusion.
  • Lung-protective ventilation (ARDSNet protocol): pH goal 7.30–7.45. If pH is 7.15–7.30, raise the rate (max 35/min). If pH is below 7.15, raise the rate to 35/min; if pH stays below 7.15, tidal volume may rise in 1 mL/kg steps until pH exceeds 7.15 (the plateau pressure target of 30 cm H₂O may then be exceeded). Above 7.45, lower the rate if possible.
  • Dialysis: refractory acidemia, toxic alcohols, salicylate, or alkalosis with kidney failure and volume overload. Involve nephrology and toxicology early.
  • Chronic CO₂ retainers: correct PaCO₂ slowly, over hours, toward the patient's baseline to avoid posthypercapnic alkalosis.

Doses are typical references. Follow local protocols, medical direction and institutional policy.

Field & Transport Considerations

Point-of-care testing

Cartridge analyzers measure pH and PaCO₂, then calculate HCO₃⁻. Respect the cartridge temperature and hematocrit limits, and trend on one device. For pH, a venous gas will do in most patients who are not in shock. EtCO₂ on the waveform tracks ventilation. It says nothing direct about pH. Pair it with one blood gas, because the PaCO₂–EtCO₂ gap widens with shock, PE and lung disease.

Before and during interfacility transport

SettingConsideration
Before departure
Timed pH, PaCO₂, HCO₃⁻, lactate, K⁺ and glucose with the ventilator settings and EtCO₂ at the time of the draw
Ventilator switch
Match the sending minute ventilation, especially in metabolic acidosis; recheck a gas after changes if a POC analyzer is carried
Running infusions
Insulin, bicarbonate and K⁺ change pH and K⁺ en route: confirm drug, concentration and rate
Altitude and flight
Hypoxemia at cabin altitude drives hyperventilation in spontaneously breathing patients; keep SpO₂ at target
Redirect
Toxic alcohol, salicylate or refractory acidemia: go to a center that can start hemodialysis

Special Populations

PopulationWhat changesClinical implication
Neonates & infants
Transitional values after birth; use the lab's neonatal range
Interpret cord and first-day gases with neonatal norms, not adult cutoffs
Children & adolescents
Adult pH range. DKA per ISPAD 2022: venous pH <7.3; severe at pH <7.1
Bicarbonate is avoided in pediatric DKA except in exceptional cases; see weight-based dosing
Pregnancy
Progesterone drives ventilation: PaCO₂ near 30 mmHg, lower HCO₃⁻, pH at the upper end of normal
A PaCO₂ of 40 mmHg is relatively high in late pregnancy and may signal tiring or ventilatory failure
Older adults
No clinically meaningful difference in pH
Review diuretics and other drugs that change acid–base status
Chronic CO₂ retainers
Baseline chronic respiratory acidosis with high HCO₃⁻
Target the patient's baseline PaCO₂, not 40 mmHg
CKD / dialysis
Chronic metabolic acidosis is common as GFR falls
Compare with the patient's baseline; refractory acidemia in kidney failure needs dialysis
Hypothermia (including TTM)
Analyzer reports at 37 °C (98.6 °F); in-body pH is higher, about 0.015 per 1 °C below 37 °C
Most adult practice reads uncorrected values against 37 °C norms (alpha-stat); give the lab the patient's temperature
Pediatric arrest and newborns: Routine pediatric and newborn resuscitation does not include bicarbonate.

Interfering Factors & Pitfalls

Collection & handling

FactorEffectWhyAvoid / fix
Air bubbles
↑ (usually slight)
CO₂ escapes into the bubble; PaCO₂ falls
Expel air at once, cap, mix gently
Delay at room temperature
↓
Cells keep producing CO₂ and acid
Plastic syringe: analyze within 30 min (15 min if PaO₂ matters)
Icing a plastic syringe
PaO₂ artifact
Plastic is gas-permeable when cold
Keep plastic at room temperature; ice only glass syringes for delays
Excess liquid heparin
PaCO₂ and HCO₃⁻ falsely ↓
Dilution
Use dry-heparin blood-gas syringes; fill to the stated volume
Vigorous mixing
↓
Hemolysis
Roll and invert gently
Line sample with flush
Variable
Saline or drug contamination
Discard enough dead space before drawing

Physiologic / patient factors

FactorEffectWhyAvoid / fix
Painful or anxious puncture
↑
Hyperventilation lowers PaCO₂
Local anesthetic; calm the patient; note the breathing pattern
Venous sample read as arterial
↓ (about 0.03)
Venous blood carries more CO₂
Label the sample type; check PaO₂ and SaO₂ fit
Hypothermia
In-body pH higher than reported
Gas solubility changes with temperature
Report the temperature; follow local alpha-stat or pH-stat practice

Assay & device-specific

FactorEffectWhyAvoid / fix
Calculated HCO₃⁻
Inherits pH and PaCO₂ errors
Derived by the analyzer, not measured
Compare with serum total CO₂; trust measured if discordant
POC cartridge outside limits
Unreliable
Temperature or hematocrit outside the operating range
Follow the device limits; send to the lab

Common misinterpretations

  • "pH 7.37 is acidemia": 7.35–7.45 is normal. Read PaCO₂ and HCO₃⁻ before you name a disorder.
  • "A normal pH means a normal acid–base state": Opposing disorders can cancel each other out. Check the components and the anion gap.
  • "Compensation brought the pH to normal": It does not. A fully normal pH with abnormal components is a mixed disorder.
  • "Fix the pH and the K⁺ will follow": The K⁺ shift that comes with pH correction is unpredictable. Treat a dangerous K⁺ directly.
Do not trust an implausible gas: A gas that fails the [H⁺] check, or does not fit the patient in front of you, gets repeated before you act on it.

Physiology & Test Basics

What it measures

Blood pH is the net acid–base result. It is the negative logarithm of the free hydrogen-ion concentration. PaCO₂ is the respiratory half, bicarbonate and base excess the metabolic ones. The scale is logarithmic, so a small change in pH is a large change in [H⁺].

Physiology & regulation

  • Buffers: CO₂ + H₂O ⇌ H₂CO₃ ⇌ H⁺ + HCO₃⁻. Hemoglobin, protein and phosphate buffers also absorb acid, within seconds.
  • Lungs: move PaCO₂ within minutes.
  • Kidneys: Over days, they reclaim and regenerate HCO₃⁻ and excrete acid.
  • Potassium: Acidemia tends to push K⁺ up. Alkalemia tends to push it down.
  • Oxygen and calcium: Alkalemia shifts the oxyhemoglobin curve left, so less O₂ is released. It also lowers ionized Ca²⁺.
  • Heart: Severe acidemia blunts contractility and the response to catecholamines.

How it is measured

  • Specimen: whole blood in a pre-heparinized (dry heparin) blood-gas syringe; arterial (usually radial) or venous. Expel air, cap, mix gently, label the sample type, time, FiO₂ and temperature.
  • Method: pH electrode at 37 °C on a laboratory or point-of-care blood-gas analyzer; results in minutes.
  • Fasting: not required.

Why it matters

pH tells you how dangerous the problem is right now. Add PaCO₂ and HCO₃⁻, and you learn whether breathing or metabolism caused it. That answer decides whether you ventilate, resuscitate, give insulin or call for dialysis.

Clinical Pearls

pH first, then who did it: The pH tells you acidemia or alkalemia. The PaCO₂ or HCO₃⁻ that moved in the matching direction is the primary problem.
Fast breathing may be holding the pH up: In metabolic acidosis, Kussmaul breathing is the compensation. Take it away with sedation, a long apnea or a standard ventilator rate, and the pH can fall to arrest. Plan high minute ventilation before you intubate.
Prehospital: the breathing pattern is a blood gas: Deep, rapid breathing without hypoxia points to metabolic acidosis: DKA, sepsis, toxin. Slow, shallow breathing with a rising EtCO₂ is respiratory acidosis. Ventilate.
A venous gas is usually enough: Venous pH runs about 0.03 lower than arterial. That gap is small enough to diagnose and follow DKA. Draw an arterial sample when PaO₂ or an exact PaCO₂ matters, or when the patient is in shock.
Nursing: recheck after every ventilator change: Trend EtCO₂ continuously. Repeat the gas after any change in rate or tidal volume, after bicarbonate, or after a change in mental status.
Do not wait for the pH to fix the K⁺: The K⁺ shift that comes with the pH is hard to predict, most of all in lactic acidosis and DKA. If hyperkalemia shows ECG changes, treat it directly.
Normal pH, sick patient: A normal pH can hide an abnormal PaCO₂ or HCO₃⁻, or a high anion gap. That is a mixed disorder. Find both causes.

References

  1. Cao, D., Arens, A. M., Chow, S. L., et al. (2025). Part 10: Adult and pediatric special circumstances of resuscitation: 2025 American Heart Association guidelines for cardiopulmonary resuscitation and emergency cardiovascular care. Circulation, 152(16 suppl 2), S578–S672. https://doi.org/10.1161/CIR.0000000000001380
  2. Umpierrez, G. E., Davis, G. M., ElSayed, N. A., Fadini, G. P., Galindo, R. J., Hirsch, I. B., et al. (2024). Hyperglycemic crises in adults with diabetes: a consensus report. Diabetes Care, 47(8), 1257–1275. https://doi.org/10.2337/dci24-0032
  3. Glaser, N., Fritsch, M., Priyambada, L., Rewers, A., Cherubini, V., Estrada, S., Wolfsdorf, J. I., & Codner, E. (2022). ISPAD Clinical Practice Consensus Guidelines 2022: Diabetic ketoacidosis and hyperglycemic hyperosmolar state. Pediatric Diabetes, 23(7), 835–856. https://doi.org/10.1111/pedi.13406
  4. Evans, L., Rhodes, A., Alhazzani, W., Antonelli, M., Coopersmith, C. M., French, C., et al. (2021). Surviving Sepsis Campaign: international guidelines for management of sepsis and septic shock 2021. Intensive Care Medicine, 47(11), 1181–1247. https://doi.org/10.1007/s00134-021-06506-y
  5. Global Initiative for Chronic Obstructive Lung Disease. (2026). Global strategy for prevention, diagnosis and management of COPD: 2026 report. https://goldcopd.org/wp-content/uploads/2026/01/GOLD-REPORT-2026-v1.3-8Dec2025_WMV2.pdf
  6. Acute Respiratory Distress Syndrome Network. (2008). Ventilator protocol: lung-protective ventilation card (July 2008; accessed 2026-10-14). https://www.ardsnet.org/files/ventilator_protocol_2008-07.pdf
  7. International Medication Systems, Limited. (current label, accessed 2026-10-14). Naloxone hydrochloride injection, solution: prescribing information. DailyMed. https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=236349ef-2cb5-47ca-a3a5-99534c3a4996
  8. Hikma Pharmaceuticals USA Inc. (current label, accessed 2026-10-14). Acetazolamide for injection: prescribing information. DailyMed. https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=85975cf0-1286-4af9-ac7f-94bb2483bf09

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